Publikasjoner
NIBIOs ansatte publiserer flere hundre vitenskapelige artikler og forskningsrapporter hvert år. Her finner du referanser og lenker til publikasjoner og andre forsknings- og formidlingsaktiviteter. Samlingen oppdateres løpende med både nytt og historisk materiale. For mer informasjon om NIBIOs publikasjoner, besøk NIBIOs bibliotek.
2015
Forfattere
Hanne Kathrine Sjølie Hans Asbjørn K. Sørlie Bjørn Tveite Birger SolbergSammendrag
This study compares the responses of two Swedish 5-year predictive stand-level functions with the observed responses in 721 fertilization experiment plots in Norway fertilized with nitrogen (N). All plots are single-species consisting of Norway spruce (Picea abies (L.) H. Karst.) or Scots pine (Pinus sylvestris L.) fertilized with ammonium nitrate (AN) or urea. The correlations between the observed and the two predicted responses were 0.34–0.40 for all plots taken together. One response function performed well on average, but underestimated the response in pine plots and overestimated the response in spruce plots. The second function overpredicted the response on the full dataset, in spruce plots and old forest, but performed well in pine plots. Both functions overestimated the growth response in high-productive plots. Higher N deposition in Norway than in Sweden may count for parts of the deviations. Testing of fertilization functions on new datasets is rare, but important part of the evaluation of functions. As the functions are not well fit for predicting the growth response in spruce and high-productive plots in our sample, new functions that include N deposition are welcome.
Sammendrag
© The Author(s) 2015. This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.
Sammendrag
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Forfattere
Gisela Lüscher Philippe Jeanneret Manuel K. Schneider Andrew Hector Michaela Arndorfer Katalin Balázs András Báldi Debra Bailey Jean-Philippe Choisis Peter Dennis Sebastian Eiter Zoltán Elek Wendy Fjellstad Phillipa K. Gillingham Maximilian Kainz Anikó Kovács-Hostyánszki Kurt-Jürgen Hülsbergen Maurizio Guido Paoletti Susanne Papaja-Hülsbergen Jean-Pierre Sarthou Norman Siebrecht Sebastian Wolfrum Felix HerzogSammendrag
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Sammendrag
In the context of reducing CO2 emissions to the atmosphere, chemical absorption with amines is emerging as the most advanced technology for post-combustion CO2 capture from exhaust gases of fossil fuel power plants. Despite amine solvent recycling during the capture process, degradation products are formed and released into the environment, among them aliphatic nitramines, for which the environmental impact is unknown. In this study, we determined the acute and chronic toxicity of two nitramines identified as important transformation products of amine-based carbon capture, dimethylnitramine and ethanolnitramine, using a multi-trophic suite of bioassays. The results were then used to produce the first environmental risk assessment for the marine ecosystem. In addition, the in vivo genotoxicity of nitramines was studied by adapting the comet assay to cells from experimentally exposed fish. Overall, based on the whole organism bioassays, the toxicity of both nitramines was considered to be low. The most sensitive response to both compounds was found in oysters, and dimethylnitramine was consistently more toxic than ethanolnitramine in all bioassays. The Predicted No Effect Concentrations for dimethylnitramine and ethanolnitramine were 0.08 and 0.18 mg/L, respectively. The genotoxicity assessment revealed contrasting results to the whole organism bioassays, with ethanolnitramine found to be more genotoxic than dimethylnitramine by three orders of magnitude. At the lowest ethanolnitramine concentration (1 mg/L), 84% DNA damage was observed, whereas 100 mg/L dimethylnitramine was required to cause 37% DNA damage. The mechanisms of genotoxicity were also shown to differ between the two compounds, with oxidation of the DNA bases responsible for over 90% of the genotoxicity of dimethylnitramine, whereas DNA strand breaks and alkali-labile sites were responsible for over 90% of the genotoxicity of ethanolnitramine. Fish exposed to > 3 mg/L ethanolnitramine had virtually no DNA left in their red blood cells.
Forfattere
Peter Waldner Anne Thimonier Elisabeth Graf Pannatier Sophia Etzold Maria Schmitt Aldo Marchetto Pasi Rautio Kirsti Derome Tiina M. Nieminen Seppo Nevalainen Antti-Jussi Lindroos Päivi Merilä Georg Kindermann Markus Neumann Nathalie Cools Bruno de Vos Peter Roskams Arne Verstraeten Karin Hansen Gunilla Pihl Karlsson Hans-Peter Dietrich Stephan Raspe Richard Fischer Martin Lorenz Susanne Iost Oliver Granke Tanja G. M. Sanders Alexa Michel Hans-Dieter Nagel Thomas Scheuschner Primož Simončič Klaus von Wilpert Henning Meesenburg Stefan Fleck Sue Benham Elena Vanguelova Nicholas Clarke Morten Ingerslev Lars Vesterdal Per Gundersen Inge Stupak Mathieu Jonard Nenad Potočić Mayte MinayaSammendrag
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Forfattere
E.J. Ahrenfeldt B.K. Klatt J. Arildsen Nina Trandem G.K.S. Andersson T. Tscharntke H.G. Smith L. SigsgaardSammendrag
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